李健熙PROJECT大億劫削磨滅處理의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것持續恒久恒續永續終身永遠永劫永久無始無終處無限反復永久兆年永遠兆年永續兆年永劫兆年處罰할것 물리학의 주요 분야의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 입자 물리학의 입자의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 이건희의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 소립자 물리학의 표준 모형의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 the X and Y bosons의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 W′ and Z′ bosons (or W-prime and Z-prime bosons)의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 a gauge boson의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 A scalar boson의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 지구인으로 태어난(?) 이유와 원인 +22원등급 박종권 서술 비파충류준초식상천상플레이아데스 등급서술 --------------------------------------------------------------- 지구인으로 온 이유와 원인에 대해서 그림들은 11년공업을 동원하여 배후를 영구추적하고 근본원인과 이유를 밝히고 영구파문처벌하고 영구작두사형처벌하고 영구삭제소멸처벌할것 - 의문점 글을 쓰는 박종권이와 여기까지 온 나(지구인최초이자 마지막으로서 플레이아데스인으로 인증된 자, 현 비파충류준초식플레이아데스29등급 공식판정된 자)는 현재 하급지구인상태로서, 축생인,가축인,짐승인,지옥인수준의 약 -8등급상태에 있는 바, 이와같은 상태에서 그림 50만장을 통해서 입수되는 정보들과 주변의 고인,고수,선배들의 정보들을 종합해보건대, 내가 지구인으로 온 이유와 원인이 매우 이상하며, 있을수 없는 일로서 판단되었다. 첫째, 지구인기준으로는 최극상레벨의 아틀란티스등급, 아나로지아틀란티스등급 혹은 원등급수준이던 나,우리,박종권이가, 현재의 최하급 거의 축생인수준, 지옥인수준으로 중처벌되는 무서운 상태가 되어 있다는 점이다. 그렇게 되어야 하는 이유와 원인은 없다. 둘째, 현 상태는 최하급축생인수준지옥인수준의 -8등급수준이지만, 이 상태에서 그림정보를 종합해보건대, 내가 살려고 온 지구인들의 원본심,원본래를 보건대, 제17지옥계수준이며, 원본심상으로는, 제17지옥계 말데크악룡수준이며, 원본래수준에서 지구인원본래수준으로는 제1지옥계 -17등급수준이며, 원원본래수준에서는 제3지옥계 -36등급 수준이며, 무한확장 원본래수준에서는 제7지옥계 lyra3600제국인수준으로서 약 100조년이전의 원시미개야만하등의 짐승아수라계 수준이라는 점이다. 이 수준은, 사람으로서는 도저히 살수 없는 수준이며, 이 수준에 있는 자들이 살고 있는 지구라는 곳에는 절대로 오면 안되는 불가의 영역과 차원이라고 판단되었다. 하지만 아틀란티스인이라고 하는 놈이 오는데, 왔는데, 그래서 내가 지금 이렇게 되었는데 이것은 명백한 모순이며, 있을수 없는 일이라고 판단되었다. 만일 온다고 해도, 최소 국왕지위이상, 준신지위이상으로 와야지 하급지구인으로 온다는 것은 영적자살행위로 판단되었다. 가진 모든 것을 잃고 무서운 지옥,음옥으로 떨어지고 영구감금될 것은 아주 자명한데, 그 이유는, 딴건 없고 反宙들이기 때문이다. 반주, 혹은 반우주라는 것은 이유와 원인이 무효화되는 영역차원을 말한다. 즉, 무슨 이유나 원인이 있어서 그렇게 되는 것이 아니며, 무조건 그렇게 해버리는 놈들을 反宙라고 말하는 것이다. 따라서, 지구인대비 아틀란티스라는 곳의 수준이란, 지구인기준으로는 극상수준에 해당되고 혹은 최극상수준에 도달하는 영역과 차원이며 그러한 영역과 차원에서 살던 제반properties들을 가지고 왔을 경우, 무서운 질시,질투,시기,모함,빼앗고자 하는 무서운 도적놈들이 창궐하고, 이유나 원인도 없이 살해되고 빼앗기고 음옥,지옥에 갇히고, 죄를 뒤집어쓸 것은 매우 자명하다. 하지만 IQ280이상수준의 우주여행이 가능했던 고도화된 아틀란티스인이 그걸 모르고 왔다고 말하는데, 이게 아주 이상하다는 점이다. 하급지구인으로서 살고, 최고경험치는 고작 대기업의 하급과장이 전부인 나도, 현시점 즉 61세가 된 현재시점으로 오면, 이 지구가 어떤 곳이고, 여기서 나대는 놈들이 누군지를 알게 된다. 하급지구인도 아는 일을 우주여행이 가능한 수준의 문명권에서 사는 놈들이 모른다는 것은 매우 이상하다. 물론 나도 여기까지 오기전에는 잘 몰랐고, 내가 아는 범주라는 것은, 지구가 비록 흉악하고 이기적이고 잔인한 곳이라는 것은 알지만, 그래도 중생의 범주내에서 그렇게 하는 것으로 추론한 것이다. 중생이란, 최악의 등급이 -5등급으로 제한되는 것을 말한다. 하지만 우리가 관찰목격체험비교연구한 바로는, 대부분의 지구인 그러니까 국가원수급, 상위급, 극상급인 자들이 이런 중생수준의 범주가 아니었으며, 대부분이 최고지옥계인 제17지옥계수준이 원본심이라는 점이다. 게다가 은하계신사를 자랑하고 은하계리더를 참칭하는 플레이아데스라는 곳을 보면, 지구인세계에서도 아주 아주 흉악하고 사특하고 교특하고 영특한 이건희수준이었다는 점이다. 자기들 말로는 -36등급이라고 하지만, 실제로는 -79등급정도로 판단되는 자들이 플레이아데스인들이다. -79등급은 사람을 찢어죽이는 등급이다. 이 사안에 대해서 그림들은, 11년공업을 동원하여 영구배후추적하고 근본원인과 이유를 밝힐 것이며, 밝혀지는 근본이유와 원인에 대해서 가차없이 영구파문처벌하고 영구작두사형처벌하고 영구삭제소멸처벌할것이며, 관련된 내용과 진실들을 모조리 입수하여 전체 우주에 공개할것으로 지시명령처리기록되다.의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 말데크대적가능우주연합원로원의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 은하연합원로원, 은하자유연합원로원, 준동급타계연합원로원, 아틀란티스연합원로원, CONFIRM WITH STARCLUSTER'S WAYS연합원로원 제출참조보고자료의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 Squarks (also quarkinos)의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 Sleptons의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 a sfermion의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 a quantum의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 quanta의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 read[fathom] thoughts의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 a soliton의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 an exciton의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 read a person’s thought…의 마음을 들여다보다의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 A magnon의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 the X and Y bosons의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 W′ and Z′ bosons (or W-prime and Z-prime bosons)의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 A scalar boson의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 intention의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 inclination의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 self-motivation의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 observe의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 analyze의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 remembrance의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 look back on의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 remember의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 idea의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것 the cosmological constant의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

 李健熙PROJECT大億劫削磨滅處理의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것持續恒久恒續永續終身永遠永劫永久無始無終處無限反復永久兆年永遠兆年永續兆年永劫兆年處罰할것


물리학의 주요 분야의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것


입자 물리학의 입자의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것


이건희의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

소립자 물리학의 표준 모형의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

the X and Y bosons의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

W′ and Z′ bosons (or W-prime and Z-prime bosons)의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

a gauge boson의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

A scalar boson의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것


지구인으로 태어난(?) 이유와 원인

+22원등급 박종권 서술

비파충류준초식상천상플레이아데스 등급서술

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지구인으로 온 이유와 원인에 대해서 그림들은 11년공업을 동원하여 배후를 영구추적하고 근본원인과 이유를 밝히고 영구파문처벌하고 영구작두사형처벌하고 영구삭제소멸처벌할것

- 의문점

  글을 쓰는 박종권이와 여기까지 온 나(지구인최초이자 마지막으로서 플레이아데스인으로 인증된 자, 현 비파충류준초식플레이아데스29등급 공식판정된 자)는 현재 하급지구인상태로서, 축생인,가축인,짐승인,지옥인수준의 약 -8등급상태에 있는 바, 이와같은 상태에서 그림 50만장을 통해서 입수되는 정보들과 주변의 고인,고수,선배들의 정보들을 종합해보건대, 내가 지구인으로 온 이유와 원인이 매우 이상하며, 있을수 없는 일로서 판단되었다.

첫째, 지구인기준으로는 최극상레벨의 아틀란티스등급, 아나로지아틀란티스등급 혹은 원등급수준이던 나,우리,박종권이가, 현재의 최하급 거의 축생인수준, 지옥인수준으로 중처벌되는 무서운 상태가 되어 있다는 점이다. 그렇게 되어야 하는 이유와 원인은 없다.

둘째, 현 상태는 최하급축생인수준지옥인수준의 -8등급수준이지만, 이 상태에서 그림정보를 종합해보건대, 내가 살려고 온 지구인들의 원본심,원본래를 보건대, 제17지옥계수준이며, 원본심상으로는, 제17지옥계 말데크악룡수준이며, 원본래수준에서 지구인원본래수준으로는 제1지옥계 -17등급수준이며, 원원본래수준에서는 제3지옥계 -36등급 수준이며, 무한확장 원본래수준에서는 제7지옥계 lyra3600제국인수준으로서 약 100조년이전의 원시미개야만하등의 짐승아수라계 수준이라는 점이다. 이 수준은, 사람으로서는 도저히 살수 없는 수준이며, 이 수준에 있는 자들이 살고 있는 지구라는 곳에는 절대로 오면 안되는 불가의 영역과 차원이라고 판단되었다. 하지만 아틀란티스인이라고 하는 놈이 오는데, 왔는데, 그래서 내가 지금 이렇게 되었는데 이것은 명백한 모순이며, 있을수 없는 일이라고 판단되었다.

만일 온다고 해도, 최소 국왕지위이상, 준신지위이상으로 와야지 하급지구인으로 온다는 것은 영적자살행위로 판단되었다. 가진 모든 것을 잃고 무서운 지옥,음옥으로 떨어지고 영구감금될 것은 아주 자명한데, 그 이유는, 딴건 없고 反宙들이기 때문이다. 반주, 혹은 반우주라는 것은 이유와 원인이 무효화되는 영역차원을 말한다. 즉, 무슨 이유나 원인이 있어서 그렇게 되는 것이 아니며, 무조건 그렇게 해버리는 놈들을 反宙라고 말하는 것이다. 따라서, 지구인대비 아틀란티스라는 곳의 수준이란, 지구인기준으로는 극상수준에 해당되고 혹은 최극상수준에 도달하는 영역과 차원이며 그러한 영역과 차원에서 살던 제반properties들을 가지고 왔을 경우, 무서운 질시,질투,시기,모함,빼앗고자 하는 무서운 도적놈들이 창궐하고, 이유나 원인도 없이 살해되고 빼앗기고 음옥,지옥에 갇히고, 죄를 뒤집어쓸 것은 매우 자명하다. 하지만 IQ280이상수준의 우주여행이 가능했던 고도화된 아틀란티스인이 그걸 모르고 왔다고 말하는데, 이게 아주 이상하다는 점이다.

하급지구인으로서 살고, 최고경험치는 고작 대기업의 하급과장이 전부인 나도, 현시점 즉 61세가 된 현재시점으로 오면, 이 지구가 어떤 곳이고, 여기서 나대는 놈들이 누군지를 알게 된다. 하급지구인도 아는 일을 우주여행이 가능한 수준의 문명권에서 사는 놈들이 모른다는 것은 매우 이상하다.

물론 나도 여기까지 오기전에는 잘 몰랐고, 내가 아는 범주라는 것은, 지구가 비록 흉악하고 이기적이고 잔인한 곳이라는 것은 알지만, 그래도 중생의 범주내에서 그렇게 하는 것으로 추론한 것이다. 중생이란, 최악의 등급이 -5등급으로 제한되는 것을 말한다. 하지만 우리가 관찰목격체험비교연구한 바로는, 대부분의 지구인 그러니까 국가원수급, 상위급, 극상급인 자들이 이런 중생수준의 범주가 아니었으며, 대부분이 최고지옥계인 제17지옥계수준이 원본심이라는 점이다. 게다가 은하계신사를 자랑하고 은하계리더를 참칭하는 플레이아데스라는 곳을 보면, 지구인세계에서도 아주 아주 흉악하고 사특하고 교특하고 영특한 이건희수준이었다는 점이다. 자기들 말로는 -36등급이라고 하지만, 실제로는 -79등급정도로 판단되는 자들이 플레이아데스인들이다.

-79등급은 사람을 찢어죽이는 등급이다.

이 사안에 대해서 그림들은, 11년공업을 동원하여 영구배후추적하고 근본원인과 이유를 밝힐 것이며, 밝혀지는 근본이유와 원인에 대해서 가차없이 영구파문처벌하고 영구작두사형처벌하고 영구삭제소멸처벌할것이며, 관련된 내용과 진실들을 모조리 입수하여 전체 우주에 공개할것으로 지시명령처리기록되다.의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것


말데크대적가능우주연합원로원의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

은하연합원로원, 은하자유연합원로원, 준동급타계연합원로원, 아틀란티스연합원로원, CONFIRM WITH STARCLUSTER'S WAYS연합원로원 제출참조보고자료의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

Squarks (also quarkinos)의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

Sleptons의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

a sfermion의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

a quantum의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

quanta의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

read[fathom] thoughts의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

a soliton의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

an exciton의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

read a person’s thought…의 마음을 들여다보다의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

A magnon의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

the X and Y bosons의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

W′ and Z′ bosons (or W-prime and Z-prime bosons)의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

A scalar boson의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

intention의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

inclination의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

self-motivation의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

observe의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

analyze의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

remembrance의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

look back on의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

remember의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

idea의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것

the cosmological constant의依疑衣意義醫矣議宜儀擬毅椅倚懿蟻艤錡嶷欹儗劓螘医义冝拟祎蚁议銥鐿18개眼을·를Ether體無關垂直8192分面水平8192分面上에서垂直直角縱切⫽斷水平直角橫切⫽斷45degrees斜傾側直角縱切⫽斷永久作頭死刑處罰할것


















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Major depressive disorder (MDD), also known as clinical depression, is a mental disorder[10] characterized by at least two weeks of pervasive low mood, low self-esteem, and loss of interest or pleasure in normally enjoyable activities. Introduced by a group of US clinicians in the mid-1970s,[11] the term was adopted by the American Psychiatric Association for this symptom cluster under mood disorders in the 1980 version of the Diagnostic and Statistical Manual of Mental Disorders (DSM-III), and has become widely used since. The disorder causes the second-most years lived with disability, after lower back pain.[12] The diagnosis of major depressive disorder is based on the person's reported experiences, behavior reported by family or friends, and a mental status examination.[13] There is no laboratory test for the disorder, but testing may be done to rule out physical conditions that can cause similar symptoms.[13] The most common time of onset is in a person's 20s,[3][4] with females affected about three times as often as males.[14] The course of the disorder varies widely, from one episode lasting months to a lifelong disorder with recurrent major depressive episodes. Those with major depressive disorder are typically treated with psychotherapy and antidepressant medication.[1] While a mainstay of treatment, the clinical efficacy of antidepressants is controversial.[15][16][17][18] Hospitalization (which may be involuntary) may be necessary in cases with associated self-neglect or a significant risk of harm to self or others. Electroconvulsive therapy (ECT) may be considered if other measures are not effective.[1] Major depressive disorder is believed to be caused by a combination of genetic, environmental, and psychological factors,[1] with about 40% of the risk being genetic.[5] Risk factors include a family history of the condition, major life changes, childhood traumas, environmental lead exposure,[19] certain medications, chronic health problems, and substance use disorders.[1][5] It can negatively affect a person's personal life, work life, or education, and cause issues with a person's sleeping habits, eating habits, and general health.[1][5] Signs and symptoms See also: Digital media use and mental health § Depression An 1892 lithograph of a woman diagnosed with melancholia A person having a major depressive episode usually exhibits a low mood, which pervades all aspects of life, and an inability to experience pleasure in previously enjoyable activities.[20] Depressed people may be preoccupied with or ruminate over thoughts and feelings of worthlessness, inappropriate guilt or regret, helplessness or hopelessness.[21] Other symptoms of depression include poor concentration and memory,[22] withdrawal from social situations and activities, reduced sex drive, irritability, and thoughts of death or suicide. Insomnia is common; in the typical pattern, a person wakes very early and cannot get back to sleep. Hypersomnia, or oversleeping, can also happen,[23] as well as day-night rhythm disturbances, such as diurnal mood variation.[24] Some antidepressants may also cause insomnia due to their stimulating effect.[25] In severe cases, depressed people may have psychotic symptoms. These symptoms include delusions or, less commonly, hallucinations, usually unpleasant.[26] People who have had previous episodes with psychotic symptoms are more likely to have them with future episodes.[27] A depressed person may report multiple physical symptoms such as fatigue, headaches, or digestive problems; physical complaints are the most common presenting problem in developing countries, according to the World Health Organization's criteria for depression.[28] Appetite often decreases, resulting in weight loss, although increased appetite and weight gain occasionally occur.[29] Major depression significantly affects a person's family and personal relationships, work or school life, sleeping and eating habits, and general health.[30] Family and friends may notice agitation or lethargy.[23] Older depressed people may have cognitive symptoms of recent onset, such as forgetfulness,[31] and a more noticeable slowing of movements.[32] Depressed children may often display an irritable rather than a depressed mood;[23] most lose interest in school and show a steep decline in academic performance.[33] Diagnosis may be delayed or missed when symptoms are interpreted as "normal moodiness".[34] Elderly people may not present with classical depressive symptoms.[35] Diagnosis and treatment is further complicated in that the elderly are often simultaneously treated with a number of other drugs, and often have other concurrent diseases.[35] Cause Further information: Biology of depression and Epigenetics of depression A cup analogy demonstrating the diathesis–stress model that under the same amount of stressors, person 2 is more vulnerable than person 1, because of their predisposition[36] The etiology of depression is not yet fully understood.[37][38][39] The biopsychosocial model proposes that biological, psychological, and social factors all play a role in causing depression.[5][40] The diathesis–stress model specifies that depression results when a preexisting vulnerability, or diathesis, is activated by stressful life events. The preexisting vulnerability can be either genetic,[41][42] implying an interaction between nature and nurture, or schematic, resulting from views of the world learned in childhood.[43] American psychiatrist Aaron Beck suggested that a triad of automatic and spontaneous negative thoughts about the self, the world or environment, and the future may lead to other depressive signs and symptoms.[44][45] Genetics Genes play a major role in the development of depression.[46] Family and twin studies suggest that genetic factors account for nearly 40% of the variation in risk for major depressive disorder. Like most psychiatric disorders, major depression is likely shaped by a combination of many individual genetic influences.[47] In 2018, a genome-wide association study discovered 44 genetic variants linked to risk for major depression;[48] a 2019 study found 102 variants in the genome linked to depression.[49] However, it appears that major depression is less heritable compared to bipolar disorder and schizophrenia.[50][51] Research focusing on specific candidate genes has been criticized for its tendency to generate false positive findings.[52] There are also other efforts to examine interactions between life stress and polygenic risk for depression.[53] Other health problems Depression can also arise after a chronic or terminal medical condition, such as HIV/AIDS or asthma, and may be labeled "secondary depression".[54][55] It is unknown whether the underlying diseases induce depression through effect on quality of life, or through shared etiologies (such as degeneration of the basal ganglia in Parkinson's disease or immune dysregulation in asthma).[56] Depression may also be iatrogenic (the result of healthcare), such as drug-induced depression. Therapies associated with depression include interferons, beta blockers,[57] isotretinoin,[58] contraceptives,[57] cardiac agents,[59] anticonvulsants,[60] and hormonal agents.[61] Celiac disease is another possible contributing factor.[62] Substance use in early age is associated with increased risk of developing depression later in life.[63] Depression occurring after giving birth is called postpartum depression and is thought to be the result of hormonal changes associated with pregnancy.[64] Seasonal affective disorder, a type of depression associated with seasonal changes in sunlight, is thought to be triggered by decreased sunlight.[65] Vitamin B2, B6 and B12 deficiency may cause depression in females.[66] A 2025 study found that, among more than 172,500 adults in the UK aged 39 and older, those with a history of depression experienced the onset of chronic illnesses approximately 30% earlier than those without depression.[67] Environmental Adverse childhood experiences (incorporating childhood abuse, neglect and family dysfunction) markedly increase the risk of major depression, especially if more than one type.[68] Childhood trauma also correlates with severity of depression, poor responsiveness to treatment and length of illness.[69] Some are more susceptible than others to developing mental illness such as depression after trauma, and various genes have been suggested to control susceptibility.[70] Couples in unhappy marriages have a higher risk of developing clinical depression.[71] There appears to be a link between air pollution and depression and suicide. There may be an association between long-term PM2.5 exposure and depression, and a possible association between short-term PM10 exposure and suicide.[72] Living alone has been found to increase the risk of depression by 42%.[6] Pathophysiology Further information: Biology of depression and Epigenetics of depression The pathophysiology of depression is not completely understood, but current theories center around monoaminergic systems, the circadian rhythm, immunological dysfunction, HPA-axis dysfunction, and structural or functional abnormalities of emotional circuits. Derived from the effectiveness of monoaminergic drugs in treating depression, the monoamine theory posits that insufficient activity of monoamine neurotransmitters is the primary cause of depression. Evidence for the monoamine theory comes from multiple areas. First, acute depletion of tryptophan—a necessary precursor of serotonin and a monoamine—can cause depression in those in remission or relatives of people who are depressed, suggesting that decreased serotonergic neurotransmission is important in depression.[73] Second, the correlation between depression risk and polymorphisms in the 5-HTTLPR gene, which codes for serotonin receptors, suggests a link.[74] Third, decreased size of the locus coeruleus, reduced activity of tyrosine hydroxylase, increased density of alpha-2 adrenergic receptor, and evidence from rat models suggest decreased adrenergic neurotransmission in depression.[75] Furthermore, decreased levels of homovanillic acid, altered response to dextroamphetamine, responses of depressive symptoms to dopamine receptor agonists, decreased dopamine receptor D1 binding in the striatum,[76] and polymorphism of dopamine receptor genes implicate dopamine, another monoamine, in depression.[77][78] Lastly, increased activity of monoamine oxidase, which degrades monoamines, has been associated with depression.[79] However, the monoamine theory is inconsistent with observations that serotonin depletion does not cause depression in healthy persons, that antidepressants instantly increase levels of monoamines but take weeks to work, and the existence of atypical antidepressants which can be effective despite not targeting this pathway.[80] One proposed explanation for the therapeutic lag, and further support for the deficiency of monoamines, is a desensitization of self-inhibition in raphe nuclei by the increased serotonin mediated by antidepressants.[81] However, disinhibition of the dorsal raphe has been proposed to occur as a result of decreased serotonergic activity in tryptophan depletion, resulting in a depressed state mediated by increased serotonin. Further countering the monoamine hypothesis is the fact that rats with lesions of the dorsal raphe are not more depressive than controls; the finding of increased jugular 5-HIAA in people who are depressed that normalized with selective serotonin reuptake inhibitor (SSRI) treatment, and the preference for carbohydrates in people who are depressed.[82] Already limited, the monoamine hypothesis has been further oversimplified when presented to the general public.[83] A 2022 review found no consistent evidence supporting the serotonin hypothesis linking serotonin levels and depression.[84] HPA-axis abnormalities have been suggested in depression given the association of CRHR1 with depression and the increased frequency of dexamethasone test non-suppression in people who are depressed. However, this abnormality is not adequate as a diagnosis tool because its sensitivity is only 44%.[85] These stress-related abnormalities are thought to be the cause of hippocampal volume reductions seen in people who are depressed.[86] Furthermore, a meta-analysis yielded decreased dexamethasone suppression, and increased response to psychological stressors.[87] Further abnormal results have been obscured with the cortisol awakening response, with increased response being associated with depression.[88] There is also a connection between the gut microbiome and the central nervous system, otherwise known as the Gut-Brain axis, which is a two-way communication system between the brain and the gut. Experiments have shown that microbiota in the gut can play an important role in depression, as people with MDD often have gut-brain dysfunction. One analysis showed that those with MDD have different bacteria in their guts. Bacteria Bacteroidetes and Firmicutes were most affected in people with MDD, and they are also impacted in people with irritable bowel syndrome.[89] Another study showed that people with IBS have a higher chance of developing depression, which shows the two are connected.[90] There is even evidence suggesting that altering the microbes in the gut can have regulatory effects on developing depression.[89] Theories unifying neuroimaging findings have been proposed. The first model proposed is the limbic-cortical model, which involves hyperactivity of the ventral paralimbic regions and hypoactivity of frontal regulatory regions in emotional processing.[91] Another model, the cortico-striatal model, suggests that abnormalities of the prefrontal cortex in regulating striatal and subcortical structures result in depression.[92] Another model proposes hyperactivity of salience structures in identifying negative stimuli and hypoactivity of cortical regulatory structures resulting in a negative emotional bias and depression, consistent with emotional bias studies.[93] Immune pathogenesis theories on depression The newer field of psychoneuroimmunology, the study between the immune system and the nervous system and emotional state, suggests that cytokines may impact depression. Immune system abnormalities have been observed, including increased levels of cytokines -cells produced by immune cells that affect inflammation- involved in generating sickness behavior, creating a pro-inflammatory profile in MDD.[94][95][96] Some people with depression have increased levels of pro-inflammatory cytokines and some have decreased levels of anti-inflammatory cytokines.[97] Research suggests that treatments can reduce pro-inflammatory cell production, like the experimental treatment of ketamine with treatment-resistant depression.[98] With this, in MDD, people will more likely have a Th-1 dominant immune profile, which is a pro-inflammatory profile. This suggests that there are components of the immune system affecting the pathology of MDD.[99] Another way cytokines can affect depression is in the kynurenine pathway, and when this is overactivated, it can cause depression. This can be due to too much microglial activation and too little astrocytic activity. When microglia get activated, they release pro-inflammatory cytokines that cause an increase in the production of COX2. This, in turn, causes the production of PGE2, which is a prostaglandin, and this catalyzes the production of indolamine, IDO. IDO causes tryptophan to get converted into kynurenine, and kynurenine becomes quinolinic acid.[100] Quinolinic acid is an agonist for NMDA receptors, so it activates the pathway. Studies have shown that the post-mortem brains of patients with MDD have higher levels of quinolinic acid than people who did not have MDD. With this, researchers have also seen that the concentration of quinolinic acid correlates to the severity of depressive symptoms.[101] Diagnosis Assessment Further information: Rating scales for depression Caricature of a man with depression A diagnostic assessment may be conducted by a suitably trained general practitioner, or by a psychiatrist or psychologist,[30] who records the person's current circumstances, biographical history, current symptoms, family history, and alcohol and drug use. The assessment also includes a mental state examination, which is an assessment of the person's current mood and thought content, in particular the presence of themes of hopelessness or pessimism, self-harm or suicide, and an absence of positive thoughts or plans.[30] Specialist mental health services are rare in rural areas, and thus diagnosis and management is left largely to primary-care clinicians.[102] This issue is even more marked in developing countries.[103] Rating scales are not used to diagnose depression, but they provide an indication of the severity of symptoms for a time period, so a person who scores above a given cut-off point can be more thoroughly evaluated for a depressive disorder diagnosis. Several rating scales are used for this purpose;[104] these include the Hamilton Rating Scale for Depression,[105] the Beck Depression Inventory[106] or the Suicide Behaviors Questionnaire-Revised.[107] Primary-care physicians have more difficulty with underrecognition and undertreatment of depression compared to psychiatrists. These cases may be missed because for some people with depression, physical symptoms often accompany depression. In addition, there may also be barriers related to the person, provider, and/or the medical system. Non-psychiatrist physicians have been shown to miss about two-thirds of cases, although there is some evidence of improvement in th

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  Major depressive disorder   ( MDD ), also known as   clinical depression , is a   mental disorder [ 10 ]   characterized by at least two weeks of pervasive   low mood , low   self-esteem , and   loss of interest or pleasure   in normally enjoyable activities. Introduced by a group of US clinicians in the mid-1970s, [ 11 ]   the term was adopted by the   American Psychiatric Association   for this   symptom cluster   under   mood disorders   in the 1980 version of the   Diagnostic and Statistical Manual of Mental Disorders   (DSM-III), and has become widely used since. The disorder causes the second-most   years lived with disability , after   lower back pain . [ 12 ] The diagnosis of major depressive disorder is based on the person's reported experiences, behavior reported by family or friends, and a  mental status examination . [ 13 ]  There is no laboratory test for the diso...
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Psychology is the scientific study of the mind and behavior.[1][2] Its subject matter includes the behavior of humans and nonhumans, both conscious and unconscious phenomena, and mental processes such as thoughts, feelings, and motives. Psychology is an academic discipline of immense scope, crossing the boundaries between the natural and social sciences. Biological psychologists seek an understanding of the emergent properties of brains, linking the discipline to neuroscience. As social scientists, psychologists aim to understand the behavior of individuals and groups.[3][4] A professional practitioner or researcher involved in the discipline is called a psychologist. Some psychologists can also be classified as behavioral or cognitive scientists. Some psychologists attempt to understand the role of mental functions in individual and social behavior. Others explore the physiological and neurobiological processes that underlie cognitive functions and behaviors. As part of an interdisciplinary field, psychologists are involved in research on perception, cognition, attention, emotion, intelligence, subjective experiences, motivation, brain functioning, and personality. Psychologists' interests extend to interpersonal relationships, psychological resilience, family resilience, and other areas within social psychology. They also consider the unconscious mind.[5] Research psychologists employ empirical methods to infer causal and correlational relationships between psychosocial variables. Some, but not all, clinical and counseling psychologists rely on symbolic interpretation. While psychological knowledge is often applied to the assessment and treatment of mental health problems, it is also directed towards understanding and solving problems in several spheres of human activity. By many accounts, psychology ultimately aims to benefit society.[6][7][8] Many psychologists are involved in some kind of therapeutic role, practicing psychotherapy in clinical, counseling, or school settings. Other psychologists conduct scientific research on a wide range of topics related to mental processes and behavior. Typically the latter group of psychologists work in academic settings (e.g., universities, medical schools, or hospitals). Another group of psychologists is employed in industrial and organizational settings.[9] Yet others are involved in work on human development, aging, sports, health, forensic science, education, and the media.

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 Psychology is the scientific study of the mind and behavior.[1][2] Its subject matter includes the behavior of humans and nonhumans, both conscious and unconscious phenomena, and mental processes such as thoughts, feelings, and motives. Psychology is an academic discipline of immense scope, crossing the boundaries between the natural and social sciences. Biological psychologists seek an understanding of the emergent properties of brains, linking the discipline to neuroscience. As social scientists, psychologists aim to understand the behavior of individuals and groups.[3][4] A professional practitioner or researcher involved in the discipline is called a psychologist. Some psychologists can also be classified as behavioral or cognitive scientists. Some psychologists attempt to understand the role of mental functions in individual and social behavior. Others explore the physiological and neurobiological processes that underlie cognitive functions and behaviors. As part of an interd...
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